简介：甲磺酸去铁胺，Deferoxamine mesylate是一种铁螯合剂，可将游离铁与稳定的复合物结合，防止其发生化学反应。
沸点 | 966.9ºC at 760 mmHg |
熔点 | 148-149° |
分子式 | C26H52N6O11S |
分子量 | 752.895 |
闪点 | 538.5ºC |
精确质量 | 752.32959 |
PSA | 268.59 |
LogP | 2.989 |
外观性状 | 白色至灰白色粉末 |
蒸汽压 | 0mmHg at 25°C |
储存条件 | 2~10℃保存 |
水溶解性 | H2O: 50 mg/mL |
中文名称： | 甲磺酸去铁胺 |
中文别名： | 甲磺酸去铁胺;甲磺酸去铁敏;甲磺酸除铁灵;甲磺酸去铁胺标准品;甲磺酸去铁胺标准品(JP);去铁铵;去铁铵 USP标准品;去铁胺甲磺酸盐 EP标准品;去铁胺甲磺酸酯;十一酸甘油三酯;云铁胺甲磺酸酯;DEFEROX胺甲磺酸盐;去铁胺甲磺酸盐;去铁铵/去铁敏 |
英文名称： | Deferoxamine mesylate |
英文别名： | Deferoxamine mesylate;Deferoxamine mesylate salt;Deferoxamine (mesylate);EHNA;Deferoxamine Methanesulfonate;Desferrioxamine B mesylate;DFOM;DFOM,Deferoxamine methanesulfonate salt,Desferrioxamine mesylate salt;Deferoxamine methanesulfonate salt;Desferrioxamine mesylate salt;desferal;Prestwick_988;desferalmesylate;deferoxaminemesilate;deferoxaminebmesylate;DEFEROXAMINE MESYLATE;monomethanesulfonate(sa;desferrioxaminebmesylate;desferalmethanesulfonate |
CAS号： | 138-14-7 |
分子式： | C27H56N6O14S2 |
分子量： | 752.90 |
详细描述： | 创赛优选提供138-14-7，甲磺酸去铁胺，Deferoxamine mesylate，Medlife，上海现货。 Medlife，致力于提供高品质、高性价比小分子化合物的产品。 Medlife小分子化合物大量库存，提供超过2万种的抑制剂、激动剂、拮抗剂等产品，是药物及疾病研究的重要原料供应商。 铁螯合剂，Deferoxamine mesylate是一种铁螯合剂，可将游离铁与稳定的复合物结合，防止其发生化学反应。 查询关键词:“138-14-7，甲磺酸去铁胺，Deferoxamine mesylate，PC15129，Medlife，上海现货”。 |
[1]. Li Y, et al. Effects of deferoxamine on blood-brain barrier disruption after subarachnoid hemorrhage. PLoS One. 2017 Mar 1;12(3):e0172784
[2]. Duscher D, et al. Comparison of the Hydroxylase Inhibitor Dimethyloxalylglycine and the Iron Chelator Deferoxamine in Diabetic and Aged Wound Healing. Plast Reconstr Surg. 2017 Mar;139(3):695e-706e
[3]. Wang G, et al. In vitro assessment of deferoxamine on mesenchymal stromal cells from tumor and bone marrow. Environ Toxicol Pharmacol. 2017 Jan;49:58-64
[4]. Wahl EA, et al. VEGF released by deferoxamine preconditioned mesenchymal stem cells seeded on collagen-GAG substrates enhances neovascularization. Sci Rep. 2016 Nov 10;6:36879
[5]. Fine JM, et al. Intranasal deferoxamine engages multiple pathways to decrease memory loss in the APP/PS1 model of amyloid accumulation. Neurosci Lett. 2015 Jan 1;584:362-7
